Rachel Irengo spent the Summer of 2021 investigating how bioengineered therapeutics can synergize with pro-repair programs active after ischemic stroke, to promote tissue regeneration in the stroke cavity and thereby improve neurological recovery. To do this, she used techniques such as confocal imaging, immunostaining, data analysis, cryosectioning, and optimized protocols for inducing stroke and hydrogel injection.
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This is amazing stuff! I see where you noted that there is an increase in inflammation without the presence of the heparin nanoparticles; are the nanoparticles used to increase mobility of the VEGF protein, or does this fall under the mechanism of action which is currently unknown?
Hey, Rachel! Your research is both insightful and interesting! You mention that CLUVENA-HA encourages the pro-repair phenotype in the young adult mice; is the tracking the mouse’s movements a biomarker of this pro-repair phenotype? Do specific movements account for full functional recovery?
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